If you want others to be happy, practice compassion. If you want to be happy, practice compassion.
— The Dalai Lama
The Vienna Declaration is a statement seeking to improve community health and safety by calling for the incorporation of scientific evidence into illicit drug policies.
Scientists, health practitioners, organizations, and the public are invited to endorse this document, to bring these issues to the attention of governments and international agencies and to
illustrate that drug policy reform is a matter of urgent international significance.
Everybody is a genius. But if you judge a fish by its ability to climb a tree, it will live its whole life believing that it is stupid.
— Albert Einstein
Empirical evidence supports the efficacy of psychodynamic therapy. Effect sizes for psychodynamic
psychotherapy are as large as those reported for other therapies that have been actively promoted as
"empirically supported" and "evidence based." Additionally, patients who receive psychodynamic
therapy maintain therapeutic gains and appear to continue to improve after treatment ends. Finally, nonpsychodynamic
therapies may be effective in part because the more skilled practitioners utilize techniques
that have long been central to psychodynamic theory and practice. The perception that psychodynamic
approaches lack empirical support does not accord with available scientific evidence and may reflect
selective dissemination of research findings. [...]
Psychodynamic or psychoanalytic [the author uses those terms interchangeably] psychotherapy refers to a range of treatments based on
psychoanalytic concepts and methods that involve less frequent meetings and may be considerably briefer
than psychoanalysis proper. Session frequency is typically once or twice per week and the treatment may
be either time limited or open ended. The essence of psychodynamic therapy is exploring those aspects of
self that are not fully known, especially as they are manifested and potentially influenced in the therapy
relationship. [...]
[...] We not only know placebos "work," we know there is a hierarchy of effectiveness:
Placebo surgery works better than placebo injections
Placebo injections work better than placebo pills
Sham acupuncture treatment works better than a placebo pill
Capsules work better than tablets
Big pills work better than small
The more doses a day, the better
The more expensive, the better
The color of the pill makes a difference
Telling the patient, "This will relieve your pain" works better than saying "This might help."
Hall's article provides an excellent review of the placebo effect, which clearly plays an important role in treatment.
In Placebos Do Work: Let's Consider Why, Christopher Lane (Shyness: How normal behavior became an illness)
expresses the hope that Hall's article will return attention to the "exciting opportunities and real quandaries (medical and ethical) that the placebo effect poses", in contrast to our largely exclusive focus
on biology and genes. Lane reminds us of an assertion by Philip Newton (3.12.08): In some controversial cases, such as selective serotonin reuptake inhibitor
(SSRI) anti- depressants, placebo effects are thought to account for a major proportion of the positive effects of a drug. He reviews a controversial paper by Irving Kirsch
and Guy Sapirstein (1998), emphasizes Kirsch's comment that their data show "virtually all of the
variation in drug effect size was due to the placebo characteristics of the studies," indicating that "the placebo component of the response to medication is considerably greater than the pharmacological
effect".
For a list of all the ways technology has failed to improve the quality of life, please press three.
— Alice Kahn (b. 1943) American writer
[...] "Quality of life" is a common phrase. The majority of human endeavours are ostensibly aimed at improving quality of life, whether for the individual or the community, and the concept
ultimately informs most aspects of public policy and private enterprise.
Advancements in healthcare have been responsible for the most significant quality-of-life gains in the recent
past: that humans are (on average) living longer, and more healthily than ever, is well established. But "quality of death" is another matter. Death, although inevitable, is distressing
to contemplate and in many cultures is taboo.
Even where the issue can be openly discussed, the obligations implied by the Hippocratic oath — rightly the starting point for all curative
medicine — do not fit easily with the demands of end-of-life palliative care, where the patient's recovery is unlikely and instead the task falls to the physician (or, more often, caregiver)
to minimise suffering as death approaches. Too often such care is simply not available: according to the Worldwide Palliative Care Alliance, while more than 100m people annually would benefit
from hospice and palliative care (including family and carers who need help and assistance in caring), less than 8% of those in need access it.
Few nations, including rich ones with cutting-edge healthcare systems, incorporate palliative care strategies into their overall healthcare policy—despite the fact that in many of these
countries, increasing longevity and ageing populations mean demand for end-of-life care is likely to rise sharply. Globally, training for palliative care is rarely included in healthcare
education curricula. Institutions that specialise in giving palliative and end-of-life care are often not part of national healthcare systems, and many rely on volunteer or charitable status.
Added to this, the availability of painkilling drugs—the most basic issue in the minimisation of suffering—is woefully inadequate across much of the world, often because of concerns about
illicit use and trafficking. The result of this state of affairs is an incalculable surfeit of suffering, not just for those about to die, but also for their loved ones. Clearly, the deeper
inclusion of palliative care into broader health policy, and the improvement of standards of end-of-life care—raising the "quality of death"—will also yield significant gains for humanity's
quality of life. [...]
[...] Since July 2009, when Environmental Working Group released its groundbreaking Bottled Water Scorecard, documenting the industry's failure to disclose contaminants and other crucial facts about their
products, bottled water producers have been taking withering fire from consumer and environmental groups.
A new EWG survey of 173 unique bottled water products finds a few improvements – but still too many secrets and too much advertising hype. Overall, 18 percent of bottled waters fail to list the
source, and 32 percent disclose nothing about the treatment or purity of the water. Much of the marketing nonsense that drew ridicule last year can still be found on a number of labels.
EWG recommends that you drink filtered tap water. You'll save money, drink water that's purer than tap water and help solve the global glut of plastic bottles.
Until the federal Food and Drug Administration cracks down on water bottlers, use EWG's [online] Bottled Water Scorecard to find brands that disclose water source, treatment and quality
and that use advanced treatment methods to remove a broad range of pollutants. → Download the PDF
Tools for Personal Development An Online Journal
Homelessness and Poverty in Canada
SOCIO Homeless in Canada
Selected resources, news and statistics on housing and homelessness in Canada, with national
and provincial housing and homeless initiatives, developments and services in major cities, local Ottawa services and charities,
research and policy organizations, food banks by province, and a customized search engine for Poverty & Homelessness in Canada.
Youth Homelessness and the Role of the Family
Family Matters - Homeless youth and Eva's Initiatives "Family Reconnect" Program. York University report calls for reform in the approach to dealing with youth homelessness and emphasizes the potential role family members can play in supporting youngsters in need. (April 2011)
Winnipeg Street Health Report 2011 United Way agency partner Main Street Project interviewed 300 people who
experience homelessness in Winnipeg, and released a report recommending quick action aimed at addressing the root causes of homelessness. (6.04.11)
Death rate from tuberculosis in homeless alarmingly high: study
Years after provincial inquiry, more work is needed to improve prevention and treatment measures for people with tuberculosis. One in five homeless people with tuberculosis dies within a year of diagnosis,
according to a study led by Dr. Kamran Khan, St. Michael's Hospital. And that number remains unchanged over the last decade despite recommendations calling for greater improvements in prevention and control of
tuberculosis in homeless shelters. Toronto (16 Feb 2011)
Building Foundations: Building Futures
Ontario's Long-Term Affordable Housing Strategy (released 29.11.10), and critique from the thestar.com (30.11.10)
Homelessness Resource Center Special Journal Issues: Future of Homeless Services
A special issue of the Open Health Services and Policy Journal on "The Future of Homeless Services". Describes the services and supports needed to help
individuals and families exit homelessness and maintain housing. Nine open access articles. (2010)
Red Tent Campaign
Pivot Legal Society, the Citywide Housing Coalition and other partners launch their Olympic campaign to attract international attention to the problem of homelessness. Vancouver.
(25.01.10)
Changing Patterns for Street Involved Youth
Groundbreaking study reveals new information about the challenges and dangers facing Toronto's street-involved youth, and presents findings on how to address this growing crisis. By Public
Interest, for Yonge Street Mission and World Vision Canada (December 2009).
Predicting Homelessness Homeless Asset and Risk Tool (HART) Calgary researchers have developed a short list of
questions to help identify people at risk before they lose their homes. (11.12.09)
Youth Homelessness in Canada : The Road to Solutions.
A document that outlines solutions to youth homelessness, based on three years of research and consultation with stakeholders across Canada. Raising the Roof. (November 2009)
$134,000 per year Lifestyle
That's what one Calgary study said it costs to support a chronically
homeless person: This week a major report recommends how Edmonton should handle its own crisis.
Richard Warnica, Edmonton Journal (26.01.09)
The Winnipeg Statement: The Provincial and Territorial Road to Poverty Reduction
Ratified in principle by participants at a cross-Canada roundtable convened in Winnipeg on August 4, 2010 by the Social
Planning Council of Winnipeg and Campaign 2000 in collaboration with the Council of Canadians with Disabilities and the
Canadian Council on Social Development.
Low Income Lines, 2008-2009
Statistics Canada, Catalogue no. 75F0002M, no. 005. (17.06.10) Covers LICOs, LIMs, and Market Basket Measure (MBMs) of low income.
The Social Determinants of Health: The Canadian Facts
Juha Mikkonen and Dennis Raphael. (May 2010) This must-read report, the only one of its kind in Canada, explores the key social determinants that affect Canadians, examines their ramifications, and
suggests specific economic and social policy strategies to remediate factors which negatively affect our health and longevity.
A Living Wage for Canadian Families The New Westminster City Council voted unanimously yesterday for a living wage bylaw
based on a calculation of the hourly wage required to keep a family with two children and two working parents above the poverty line. (28.04.10)
No Action: No Progress
Canadian Feminist Alliance for International Action Report on Canada's Progress in Implementing Priority Recommendations made by the United Nations Committee on the Elimination of
Discrimination against Women in 2008. FAFIA (February 2010)
Left Behind, Parts 1-3 CBC Ideas host Paul Kennedy presents this 3-part series by Jill Eisen, who explores the rise of inequality
that began in the 1980s and has led to the concentration of income in the top 1% of the population. This is an outstanding series. Also available as podcast.
Income inequality rising in Canada: OECD report
The rise in inequality was largely due to widening disparities in labor earnings between high and low-paid workers, but also to less redistribution. Taxes and benefits reduce inequality less in
Canada than in most OECD countries [...] (5.12.11)
Food bank use skyrockets by 26% since 2008 Food Banks Canada releases HUNGERCOUNT 2011: A comprehensive report on hunger and food bank use in Canada, and recommendations for change.
News release, report, and table showing food bank use in Canada by province. Includes individual provincial reports. (1.11.11)
Ending Poverty by Investing in People — An effective and less costly solution. The National Council on Welfare releases The Dollars and Sense of Solving Poverty,
showing hard facts and figures attesting to the cost-effectiveness of investing in people rather than continue the expensive and consistently ineffective spending patterns of the past. (28.09.11)
The poor still pay more: Challenges low income families face in consuming a nutritious diet
The Institute for Competitiveness & Prosperity, in collaboration with Open Policy Ontario's John Stapleton and research consultant from Toronto Public Health, Brian Cook, releases its report recommending initiatives to help low income families overcome challenges in consuming a nutritious diet. (21.12.10)
National Council of Welfare releases the Welfare Incomes 2009 report
From the National Council of Welfare, the Welfare Incomes 2009 report looks at the situation of four family types: a lone parent with a 2-year-old child, a couple with two children
aged 10 and 15, a single person considered employable, and a single person with a disability. (13.12.10)
The worst child income inequality in the "developed" world...
A new report by UNICEF, The Children Left Behind: A League Table of Inequality in Child Well-being in the World's Rich Countries, has shown that child poverty in Canada is among the worst of developed counties, ranked 17 out of 24.
Richest 1% income shares at historic high
Report entitled The Rise of Canada's Richest 1%, by Armine Yalnizyan, CCPA, shows that Canada's richest 1% — the 246,000 privileged few whose average income is $405,000 — took almost a third (32%) of all
growth in incomes in the fastest growing decade in this generation, 1997 to 2007. (1.12.10)
2010 Report Card on Child and Family Poverty in Canada
The 2010 Report Card on Child and Family Poverty in Canada, Reduced Poverty = Better Health for All, looks at the nation's most
recent child and family poverty rate compared to 21 years ago, when Parliament unanimously resolved to end child poverty by 2000, and finds that
610,000 children (2008 LICO after-tax) and their families lived in poverty even before the recession hit... Campaign 2000 (24.11.10)
Federal Poverty Reduction Plan
Report of the Standing Committee on Human Resources, Skills and Social Development and the Status of Persons with Disabilities, Candice Hoeppner MP, Chair; November 2010, House of Commons, 40th Parliament, 3rd Session.
"The report is based on a three-year study on the federal role in addressing poverty and will be a core element in developing a national strategy to reducing poverty in Canada," says CASW president Darlene MacDonald.
HungerCount 2010 Food Banks Canada
The results of the HungerCount 2010 survey released today show food banks across Canada helped 867,948 separate individuals in March 2010, an increase of 9.2%, or more than 73,000 people, compared to March 2009. This is 28% higher than in 2008, and is the highest level of food bank use since 1997. Food Banks Canada (16.11.10)
Bad Medicine: A Judge's Struggle for Justice in a First Nations Community
John Reilly, Rocky Mountain Books (8.11.10) At a time when government is proposing new "tough on crime" legislation, Judge Reilly provides an enlightening and timely perspective. He shows us why harsher punishments for offenders don't necessarily make our societies safer, why the white justice system is failing First Nations communities, why jail time is not the cure-all answer some think it to be, and how corruption continues to plague tribal leadership.
Disability Poverty: An Unacknowledged Reality
Council of Canadians with Disabilities (28 October 2010; 1.11.10) In Ottawa, at a national forum called End Exclusion 2010, people with disabilities and their allies are challenging the myth that people with disabilities are "well taken care of in
Canadian society." Disabling Poverty/Enabling Citizenship, Delta Ottawa Hotel, Ottawa ON, November 1 & 2, 2010.
Federal Government abandons people in poverty in Canada
Campaign2000 (20.09.10). Reaction to the 27.09.10 government response to In from the Margins: A Call to Action on Poverty, Housing and Homelessness. This post includes the government response (PDF), In from the Margins (PDF), the Statement of the Winnipeg Roundtable to the Council of the Federation (4.08.10) (PDF), and related links.
Cost of Eating in BC
Released 15 December 2009 by Dietitians of Canada, BC Region and the Community Nutritionists Council of BC, this report was endorsed by 23 organizations and highlights the fact that
not all British Columbians have enough money to buy healthy food. Dietitians are calling for the provincial government to take some additional action to address poverty in this province. Accessed 2.07.10.
Do the Math Survey The Stop Community Food Centre today announced the second phase of
"Do the Math", the anti-poverty organization's campaign to highlight the failure of Ontario's current
social assistance rates to support healthy, dignified lives. (6.04.10)
A PROPOSAL FOR ODSP RULE CHANGES: 'Stupid Rules' Have Dire Consequences
This ODSP Action Coalition submission to the Ontario Social Assistance Review Advisory Council presents a number of short-term rules changes that would make a significant
difference in the lives of people who rely on ODSP benefits. The list of recommended changes is not exhaustive, but represents a start on reforming a system that offers
insufficient support to people with disabilities. (28.01.10)
Carotenoids are a class of more than 600 naturally occurring pigments synthesized by plants, algae, and photosynthetic bacteria. These richly colored molecules are the sources of the yellow, orange,
and red colors of many plants (1). Fruits and vegetables provide
most of the carotenoids in the human diet. Alpha-carotene, beta-carotene, beta-cryptoxanthin, lutein, lycopene, and zeaxanthin are the most common dietary carotenoids. Alpha-carotene, beta-carotene
and beta-cryptoxanthin are provitamin A carotenoids, meaning they can be converted by the body to retinol
(Figure 1). Lutein, lycopene, and zeaxanthin cannot be converted to retinol, so they have no
vitamin A activity (Figure 2). Carotenoids can be broadly classified into two classes,
carotenes (alpha-carotene, beta-carotene, and lycopene) and xanthophylls (beta-cryptoxanthin, lutein, and zeaxanthin). [Read More]
Natural Sources:
Carrot juice; tomato juice, sweet potatoes, dried apricots, carrots, tomatoes, spinach, cantaloupe, pumpkin and broccoli. Other good sources of carotenoids include: dandelion greens;
fenugreek greens; dark green leafy vegetables (collards and kale); dark orange vegetables (yams and squash); Spirulina and other blue green algae; pink and red grapefruits; mango; and watermelon.
[...] Therapeutic Uses:
Acne
AIDS
Aging Disorders
Alzheimer's Disease
Anti-inflammatory
Antioxidant
Cancer Prevention
Cataracts
Cellular Regeneration
Cleansing
Detoxification
Emphysema
Eyesight Disorders
Fibromyalgia
Heart Health Maintenance
HIV Infection
Immune System
Leukemia
Lupus Nephritis
Macular Degeneration
Premenstrual Syndrome (PMS)
Skin Hyper-pigmentation
Skin Problems
Stroke Prevention
Sunburn
Ulcers
Vascular Disorders
Vitamin A Deficiency (RDA=2.3-7.8mg/day)
Wrinkles
Source:Food Sources
The World's Healthiest Foods, whfoods.org. Accessed: 12 November 2011.
What foods provide carotenoids?
The orange-colored fruits and vegetables including carrots,
apricots, mangoes, squash, and
sweet potatoes contain significant amounts of beta-carotene, alpha-carotene, and beta-cryptoxanthin.
Green vegetables, especially spinach,
kale, and
collard greens, also contain beta-carotene, and are the best sources of lutein.
This antioxidant rich, triple action blend features 3 classes of phytonutrients - Carotenoids, Polyphenols and Bioflavonoids - amplified with Vitamins C and D to
support healthy energy, endurance and mental clarity.
The importance of the microbiota that colonize and comprise us
The human body is a habitat for a huge range of harmless and beneficial microbes, which may be the key to fighting disease without antibiotics.
[... In the intensive care nursery at Duke University Medical Center, neonatologist Susan] LaTuga is one of several medical researchers at Duke working with microbial ecologists to study the development of the
human microbiome — the enormous population of microbes, including bacteria, fungi, and viruses,
that live in the human body, predominantly in the gut. There are 20 times as many of these microbes as there are cells in the body, up to 200 trillion in an adult, and each of us hosts
at least 1,000 different species. Seen through the prism of the microbiome, a person is not so much an individual human body as a superorganism made up of diverse ecosystems, each teeming with microscopic creatures
that are essential to our well-being. "Our hope is that if we can understand the normal microbial communities of healthy babies, then we can manipulate unhealthy ones," LaTuga says.
The Duke study is just one of many projects begun in the past five years that use genetic sequencing to explore how the diversity of the microbiome impacts our health. Two of the largest efforts are
the Human Microbiome Project, funded by the National Institutes of Health [see "Your Microbial Menagerie", below] and the
European Union's Metagenomics of the Human Intestinal Tract. Although these groups have only just begun to publish their
findings, it is already clear that the microbiome is much more complex and very likely more critical to human health than anyone suspected. Understanding and controlling the diversity of our germs,
as opposed to assaulting them with antibiotics, could be the key to a range of future medical treatments. [...]
"The classical view of infectious disease is that a single organism invades and produces an infection," [says
Patrick Seed, a Duke pediatrician specializing in infectious disease], "But then we found that
certain diseases, like irritable bowel syndrome, seem to be caused by imbalances in the organisms that communicate with the host. So then people asked, 'Why is this not the case for many other states of human health?'"
Preliminary work by other groups, similarly made up of both biomedical researchers and microbial ecologists, suggests that imbalances in the microbiome might also be linked to allergies,
diabetes, and obesity. [...]
Microbiome studies run directly against the notion in the minds of most people — even many researchers — that microbes are linked to disease, not to health. And of course not all microorganisms are benign. Infants
in particular are susceptible to a number of diseases caused by gastrointestinal bacteria, including sepsis, chronic diarrhea, and necrotizing enterocolitis, an infection of the intestinal lining that is one
of the leading causes of death in premature babies. Antibiotics have long been the first option in fighting these dangerous microbes, but many researchers are troubled by modern medicine's heavy reliance on
them. After all, many pathogens found within the human microbiome are harmless or even beneficial. "There is Staphylococcus and E. coli in all of us, but they don't always cause problems," Jackson
says. "It's the balance that is important. A more normal population of microbes in the gut can offset the bad players." [...]
Heavy use of antibiotics can lead to antibiotic resistance, but researchers now speculate that antibiotics can also upset the balance of the microbial community, allowing disease to take
over rather than fighting it.Michael Cotten, another neonatologist on the Duke project, analyzed the duration of antibiotic therapy given to 4,039
premature babies at 19 treatment centers across the country and found that prolonged use of the drugs is associated with
increased risk of necrotizing enterocolitis and death. Antibiotics probably also prevent beneficial bacterial communities from forming in infants.
[In 2008] Stanford microbiologist David Relman published a study that illustrated the potentially devastating impact of antibiotics on the microbiome. He gave three healthy adults a five-day course of the antibiotic Cipro,
then another course six months later, and monitored the state of the microbiome after each treatment. The gut flora of all three subjects gradually recovered from the impact of
the antibiotic treatment, but never returned to their original state — they had different compositions and were less diverse. "We don't know if these differences matter to health," Relman says. "But in general,
you'd be concerned about a change." He had chosen Cipro because it has limited effectiveness against most species of bacteria in the gut, but it still affected one-third to one-half of the microbial flora in the
subjects. "Knocking out one organism could have a ripple effect on the lives of others," Relman says.
This is especially concerning given that the number of different microbial species in the intestines may be important in countering pathogens. "The greater the diversity, the lower the probability
that pathogens can invade and persist," says Richard Ostfeld, a disease ecologist at the Cary Institute of Ecosystem
Studies in New York. "If all the niches are taken up in the gut, it might be hard for them to get hold."
Jackson puts it more bluntly. "When you use antibiotics, you are essentially dropping a bomb on a microbial community, hoping that your explosion will not harm anything useful," he says. "It's like setting
a forest fire in order to control the weeds. What we're suggesting is to carefully manipulate the members of the community and the relationships between them, rather than wiping them out." [...] Read the full article
Abstract
Microbial colonization of mammals is an evolution-driven process that modulate host physiology, many of which are associated with immunity and nutrient intake. Here, we report that colonization by gut
microbiota impacts mammalian brain development and subsequent adult behavior. Using measures of motor activity and anxiety-like behavior, we demonstrate that germ free (GF) mice display increased motor
activity and reduced anxiety, compared with specific pathogen free (SPF) mice with a normal gut microbiota. This behavioral phenotype is associated with altered expression of genes known to be involved
in second messenger pathways and synaptic long-term potentiation in brain regions implicated in motor control and anxiety-like behavior. GF mice exposed to gut microbiota early in life display similar
characteristics as SPF mice, including reduced expression of PSD-95 and synaptophysin in the striatum. Hence, our results suggest that the microbial colonization process initiates signaling mechanisms
that affect neuronal circuits involved in motor control and anxiety behavior.
Early life environmental influences have a profound impact on the organism's later development, structure, and function. This phenomenon is called "developmental programming," a process whereby an environmental
factor acting during a sensitive or vulnerable developmental period exerts effects that impact on structure and function of organs that, in some cases, will persist throughout life (1). One such environmental
factor is the gut microbiota that, because of an evolutionary process, has adapted to coexist in commensal or symbiotic relationship with mammals (2). Immediately after birth, the newborn organism is rapidly
and densely populated with complex forms of indigenous microbes. This process has been shown to contribute to developmental programming of epithelial barrier function, gut homeostasis, and angiogenesis, as
well as the innate and host adaptive immune function (3, 4). Recent data indicate that gut microbiota have systemic effects on liver function (5–7), thus raising the possibility that gut microbiota can have
developmental effects in other organs elsewhere in the body.
The functional development of the mammalian brain is of particular interest because it has been shown to be susceptible to both internal and external environmental cues during perinatal life. Epidemiological
studies have indicated an association between common neurodevelopmental disorders, such as autism and schizophrenia, and microbial pathogen infections during the perinatal period (8, 9). These findings are
supported by experimental studies in rodents, demonstrating that exposure to microbial pathogens during similar developmental periods result in behavioral abnormalities, including anxiety-like behavior and
impaired cognitive function (10–12). In a recent study, it was shown that the commensal bacteria, Bifidobacteria infantis, could modulate tryptophan metabolism, suggesting that the normal gut microbiota can
influence the precursor pool for serotonin (5-HT) (13). [...]
The human superorganism is a conglomerate of mammalian and microbial cells, with the latter estimated to outnumber the former by ten to one and the microbial
genetic repertoire (microbiome) to be approximately 100-times greater than that of the human host. Given the ability of the immune response to rapidly counter
infectious agents, it is striking that such a large density of microbes can exist in a state of synergy within the human host. This is particularly true of
the distal gastrointestinal (GI) tract, which houses up to 1000 distinct bacterial species and an estimated excess of 1 × 1014 microorganisms.
An ever-increasing body of evidence implicates the GI microbiota in defining states of health and disease. Here, we review the literature in adult and
pediatric GI microbiome studies, the emerging links between microbial community structure, function, infection and disease, and the approaches to manipulate
this crucial ecosystem to improve host health.
The gastrointestinal microbiome is associated with host health status.
Structure and composition of the microbiome defines functional gene expression of the community, pathogen abundance and physiology, and the host response.
Prenatal and early postnatal microbial exposures impact immune response development and define predisposition to the development of inflammatory diseases.
Specific microbes have demonstrated roles in immune response modulation.
Manipulation of the microbiome through pro-, pre- or synbiotic supplementation may prove an alternative approach for improving host health status.
Depiction of the human body and bacteria that predominate; there are both tremendous similarities and differences among the bacterial species found at different sites.
Topographical and temporal diversity of the human skin microbiome.
Grice EA, Kong HH, Conlan S, Deming CB, Davis J, Young AC; NISC Comparative Sequencing Program, Bouffard GG, Blakesley RW, Murray PR, Green ED, Turner ML, Segre JA.
Science. 2009 May 29;324(5931):1190-2.
Your Microbial Menagerie
Two hundred trillion microscopic organisms bacteria, viruses, and fungi are swarming inside you right now. The largest collection, weighing as much as four pounds in total, clings to your gut, but
your skin also hosts more than a million microbes per square centimeter. One population thrives among the hair follicles on your scalp, while an entirely different one resides in the crook of your elbow. About 1,000
species can live in the human mouth, where different sides of the same tooth sustain distinctly different combinations of bugs.
Surprisingly little is known about these invisible communities and how they affect us. In 2007 the National Institutes of Health (NIH) launched the
Human Microbiome Project, a $115 million initiative exploring the bugs that exist in the human body, whether people all share a core population of such
organisms, and how changes in microbial ecosystems influence human health and disease. In 2009 NIH geneticist Julie Segre published a study showing that
physiologically comparable parts of the body host similar microbial ecologies, whereas contrasting areas — say sweaty
underarms and dry forearms — have drastically different communities. "My scalp community is much more similar to your scalp than to my own back. That's because bacteria thrive in particular environments," Segre says. For
instance, she notes, the face is ideal for Propionibacterium acnes, a bug that thrives on the oily, waxy remains of dead cells. People often associate P. acnes with acne problems, but it also breaks down
oils into a natural moisturizer for the skin. [...] —Amy Barth
In 2003 Terry Wahls MD was diagnosed with secondary progressive multiple sclerosis and soon became dependent upon a tilt-recline wheelchair. After developing and using the Wahls Protocol™, she is now able to walk
through the hospital and commute to work by bicycle. She now uses intensive directed nutrition in her primary care and traumatic brain injury clinics. Dr. Wahls is the lead scientist in a clinical trial testing
her protocol in others with progressive MS.
Eating the Wahls Way™
Here are three simple food rules to follow to ensure you will have enough building blocks on hand for optimal function of your brain and reduced risk of excessive inflammation.
Eat 9 cups of vegetables and fruit (3 green, 3 sulfur, 3 color) to ensure you have enough B vitamins, minerals (sulfur, iodine, magnesium), antioxidants, and essential fats through food (greens, seafood, grass fed meat, game, wild fish, flax, walnuts)
Reduce food allergy risk (go gluten-free and dairy-free)
Eat organic, locally grown foods and grow more of your own[...]
In the fall of 2007, I had an important epiphany. What if I redesigned my diet so that I was getting those important brain nutrients not from supplements but from the foods I ate? It took more time to create
this new diet, intensive directed nutrition, which I designed to provide optimal nutrition for my brain. At that time, I also learned about neuromuscular electrical stimulation and convinced my physical therapist
to give me a test session. It hurt, a lot, but I also felt euphoric when it was finished, likely because of the endorphins my body released in response to the electrical stimulation. In December 2007, I began my
intensive directed nutrition along with a program of progressive exercise, electrical stimulation, and daily meditation. The results stunned my physician, my family and me: within a year, I was able to walk through
the hospital without a cane and even complete an 18-mile bicycle tour. [Read More]
Multiple studies have shown the way to improve health and minimize chronic health problems, including mental problems, is by eating more vegetables. The studies that added vitamins or supplements and did not
change the eating pattern have much less benefit than the studies that relied on food. For that reason, I advocate that everyone eat more greens, more sulfur and more color.
Dr. Terry Wahls links micronutrient starvation to the epidemics of chronic disease that are overtaking modern society. She explains the key roles mitochondria play in maintaining a healthy brain and body. Americans
are eating so poorly, something we all know to be true, that the majority of Americans are missing key building blocks that are needed for brain cells to be healthy. The result is an epidemic of depression, aggression,
multiple sclerosis and early dementia. She then teaches you how to eat for healthy mitochondria, a healthy brain and a healthy body in language that is clear and concise, even for those without a science background.
[...] Dr. Wahls explains basic brain biology in simple terms. She tells us what vitamin, mineral and essential fat building blocks are needed by the mitochondria and other key structures in the brain [and]
explains what foods are good sources for those key nutrients. Over a hundred recipes are provided to help get you started on this new way of eating. [...]
Large Print Edition; Apr 01 2010; ISBN/EAN13: 0982175027 / 9780982175026; 364 pages; US Trade Paper; List Price: $40.50
The rise of Internet search engines like Google has changed the way our brain remembers information, according to research by Columbia University psychologist Betsy Sparrow published July 14 in Science.
"Since the advent of search engines, we are reorganizing the way we remember things," said Sparrow. "Our brains rely on the Internet for memory in much the same way they rely on the memory of a friend,
family member or co-worker. We remember less through knowing information itself than by knowing where the information can be found."
Sparrow's research reveals that we forget things we are confident we can find on the Internet. We are more likely to remember things we think are not available online. And we are better able to remember
where to find something on the Internet than we are at remembering the information itself. This is believed to be the first research of its kind into the impact of search engines on human memory organization. [...]
[...] For me, as for others, the Net is becoming a universal medium, the conduit for most of the information that flows through my eyes and ears and into my mind. The advantages of having immediate access to such an
incredibly rich store of information are many, and they've been widely described and duly applauded. "The perfect recall of silicon memory," Wired's Clive Thompson
has written, "can be an enormous boon to thinking." But that boon comes at a price. As the media theorist
Marshall McLuhan pointed out in the 1960s, media are not just passive channels of information. They supply the stuff of thought, but
they also shape the process of thought. And what the Net seems to be doing is chipping away my capacity for concentration and contemplation. My mind now expects to take in information the way the Net
distributes it: in a swiftly moving stream of particles. Once I was a scuba diver in the sea of words. Now I zip along the surface like a guy on a Jet Ski.
I'm not the only one. When I mention my troubles with reading to friends and acquaintances — literary types, most of them — many say they're having similar experiences. The more they use the Web, the more they
have to fight to stay focused on long pieces of writing. Some of the bloggers I follow have also begun mentioning the phenomenon. Scott Karp, who writes a blog
about online media, recently confessed that he has stopped reading books altogether. "I was a lit major in college, and used to be [a] voracious book reader," he wrote. "What happened?" He speculates on the
answer: "What if I do all my reading on the web not so much because the way I read has changed, i.e. I'm just seeking convenience, but because the way I THINK has changed?"
Bruce Friedman, who blogs regularly about the use of computers in medicine, also has described how the Internet has altered his mental habits. "I
now have almost totally lost the ability to read and absorb a longish article on the web or in print," he wrote earlier this year. A pathologist who has long been on the faculty of the University of Michigan
Medical School, Friedman elaborated on his comment in a telephone conversation with me. His thinking, he said, has taken on a "staccato" quality, reflecting the way he quickly scans short passages of text
from many sources online. "I can't read War and Peace anymore," he admitted. "I've
lost the ability to do that. Even a blog post of more than three or four paragraphs is too much to absorb. I skim it."
Anecdotes alone don't prove much. And we still await the long-term neurological and psychological experiments that will provide a definitive picture of how Internet use affects cognition. But a recently
published study of online research habits [Information Behaviour of the Researcher of the Future (18 October 2007)], conducted by scholars from University College London, suggests that we may well be in the
midst of a sea change in the way we read and think. As part of the five-year research program, the scholars examined computer logs documenting the behavior of visitors to two popular research sites, one
operated by the British Library and one by a U.K. educational consortium, that provide access to journal articles, e-books, and other sources of written information. They found that people using the sites
exhibited "a form of skimming activity," hopping from one source to another and rarely returning to any source they'd already visited. They typically read no more than one or two pages of an article or book
before they would "bounce" out to another site. Sometimes they'd save a long article, but there's no evidence that they ever went back and actually read it. The authors of the study report:
It is clear that users are not reading online in the traditional sense; indeed there are signs that new forms of "reading" are emerging as users "power browse" horizontally through titles, contents pages and
abstracts going for quick wins. It almost seems that they go online to avoid reading in the traditional sense.
Thanks to the ubiquity of text on the Internet, not to mention the popularity of text-messaging on cell phones, we may well be reading more today than we did in the 1970s or 1980s, when television was our medium
of choice. But it's a different kind of reading, and behind it lies a different kind of thinking—perhaps even a new sense of the self. "We are not only what we read," says Maryanne Wolf, a developmental
psychologist at Tufts University and the author of Proust and the Squid: The Story and Science of the Reading Brain. "We
are how we read." Wolf worries that the style of reading promoted by the Net, a style that puts "efficiency" and "immediacy" above all else, may be weakening our capacity for the kind of deep reading that
emerged when an earlier technology, the printing press, made long and complex works of prose commonplace. When we read online, she says, we tend to become "mere decoders of information." Our ability to interpret
text, to make the rich mental connections that form when we read deeply and without distraction, remains largely disengaged.
Reading, explains Wolf, is not an instinctive skill for human beings. It's not etched into our genes the way speech is. We have to teach our minds how to translate the symbolic characters we see into the language
we understand. And the media or other technologies we use in learning and practicing the craft of reading play an important part in shaping the neural circuits inside our brains. Experiments demonstrate that readers
of ideograms, such as the Chinese, develop a mental circuitry for reading that is very different from the circuitry found in those of us whose written language employs an alphabet. The variations extend across many
regions of the brain, including those that govern such essential cognitive functions as memory and the interpretation of visual and auditory stimuli. We can expect as well that the circuits woven by our use of the
Net will be different from those woven by our reading of books and other printed works. [...] → Read the whole article
Within a single generation, digital media and the World Wide Web have transformed virtually every aspect of modern culture, from the way we learn and work to the ways in which we socialize and even conduct war. But
is the technology moving faster than we can adapt to it? And is our 24/7 wired world causing us to lose as much as we've gained?
In Digital Nation: Life on the Virtual Frontier, FRONTLINE presents an in-depth exploration of what it means to be human in a 21st-century digital world. Continuing a line of investigation she began with the 2008
FRONTLINE report Growing Up Online, award-winning producer
Rachel Dretzin embarks on a journey to understand the
implications of living in a world consumed by technology and the impact that this constant connectivity may have on future generations. "I'm amazed at the things my kids
are able to do online, but I'm also a little bit panicked when I realize that no one seems to know where all this technology is taking us, or its long-term effects," says Dretzin. [...] Read More
Summary
The advent of the Internet, with sophisticated algorithmic search engines, has made accessing information as easy as
lifting a finger. No longer do we have to make costly efforts to find the things we want. We can "Google" the
old classmate, find articles online, or look up the actor who was on the tip of our tongue. The results of four
studies suggest that when faced with difficult questions, people are primed to think about computers and that
when people expect to have future access to information, they have lower rates of recall of the information itself
and enhanced recall instead for where to access it. The Internet has become a primary form of external or
transactive memory, where information is stored collectively outside ourselves.
How do people remember things in relationships and groups? Each person doesn't need to remember everything the group needs to know, after all, if each person merely stores
in memory information about who is likely to have a particular item in the future. This capacity for remembering who knows what is the key to transactive memory as introduced by
Wegner, Giuliano, and Hertel (1985) and Wegner (1986). Transactive
memory suggests an analysis not only of how couples and families in close relationships coordinate memory and tasks in the home, but how larger groups and organizations come to develop "group minds," memory systems
that are more complex and potentially more effective than those of any of the individuals that comprise them.
Wegner TG, Wegner DM. "Transactive memory." In ASR Manstead & M Hewstone (Eds.), The Blackwell Encyclopedia of Social Psychology. (1995:654-656)
Oxford: Blackwell.
[...] For we shall be cyborgs not in the merely superficial sense of combining flesh and wires but in the more profound sense of being human-technology symbionts: thinking and
reasoning systems whose minds and selves are spread across biological brain and nonbiological circuitry. [...] Certainly I don't think this tendency toward cognitive
hybridization is a modern development. Rather, it is an aspect of our humanity, which is as basic and ancient as the use of speech and which has been expanding its territory
ever since. We see some of the "cognitive fossil trail" of the cyborg trait in the historical procession of potent cognitive technologies that begins with speech and counting,
morphs first into wriitten text and numerals, then into early printing (without moveable typefaces), on to the revolutions of moveable typefaces and the printing press, and most
recently to the digital encodings that bring text, sound, and image into a uniform and widely transmissible format. Such technologies, once up and running in the various
appliances and institutions that surround us, do far more than merely allow for external storage and transmission of ideas. They constitute [...] a cascade of "mindware upgrades":
cognitive upheavals in which the effective architecture of the human mind is altered and transformed. [...]
[... T]he old puzzle, the mind-body problem, really involves a hidden third party. It is the mind-body-scaffolding problem. It is the problem of understanding how human thought and
reason is born out of looping interactions between material brains, material bodies, and complex cultural and technological environments. We create these supportive envioironments,
but they create us too. We exist, as the thinking beings we are, only thanks to a baffling dance of brains, bodies, and cuultural and technological scaffolsing. Understanding this
evolutionarily novel arrangement is crucial for our science, our morals, and our self-image both as persons and as a species.
Food sources of vitamin D include fatty fish such as salmon.
The Institute of Medicine released the long-awaited Dietary Reference Intakes for Calcium and Vitamin D on 30 November 2010, detailing new guidelines by the Food and Nutrition Board. Focused on bone health, the report asserts
that there is insufficient evidence vitamin D helps protect against autoimmune diseases, diabetes, heart disease, cancer, and others diseases. It sets 400 IU/day as the Estimated Average Requirement (EAR) for adults, 600 IU/day
as the Recommended Dietary Allowance (RDA) for adults, and 800 IU/day as the appropriate amount for those aged 71+.
In Canada, several provinces have now delisted serum 25(OH)D [25-hydroxyvitamin D] testing because the demand and associated costs have risen dramatically. Health insurance will cover only certain conditions, making it
difficult for physicians and patients to monitor treatment and proactive regimens with respect to other conditions. The argument is that this delisting is "evidence-based", but researchers are all too aware that evidence
of the importance of Vitamin D, with respect to both treatment and prevention, is already compelling and growing rapidly.
What's all buzz about with respect to Vitamin D? Are the new guidelines too low? Should you take a Vitamin D supplement? If so, how much?
Nancy Piccone, Life Extension Magazine, October 2011.
The Silent Epidemic of Iodine Deficiency:
[...] Your thyroid gland is located in the front of your neck, just below your voice box. The thyroid produces two forms of thyroid hormone, both of which are derived from the amino acid tyrosine and several
atoms of iodine. Thyroid hormones control your body’s metabolism, regulating everything from body temperature and heart rate to glucose consumption and even blood lipid levels.27-29
Too much thyroid hormone (hyperthyroidism) results in an excessively high metabolic rate. People with this condition have rapid heart rates and often palpitations,29 excessive sweating, and may feel much
warmer than other people do, even in a cool room. In extreme cases they may lose weight and experience muscle weakness.
Too little thyroid hormone (hypothyroidism) results in just the opposite set of symptoms: a slower than normal heart rate,29 a chronic feeling of being cold, constipation, unexplained weight gain, dry skin,
hair loss or coarse dry hair, weakness, muscle aches, depression, and fatigue.30,31 In extreme cases, people with low thyroid function experience cognitive decline,32 and babies born to mothers with inadequate iodine
levels are at high risk for a unique form of mental retardation known as cretinism. Cognitive impairment caused by low thyroid function is reversible with iodine or thyroid hormone supplementation.32,33 [...]
Fungi are closely related to us in phylogenetic terms and, for at least four millenia in Asia, they have been used for medicinal purposes. Mushrooms and the mycelium contain an array of active
contituents, including steroids, lactones, alkaloids, polyssacharides and triterpenes. Some mushrooms have been shown to have antibacterial, antiviral, immunomodulating, adaptogenic, and antitumor
effects. This page presents three amazing videos and a brief introduction to the mycelium, then focuses on a more in-depth examination of the adaptogenic and therapeutic uses of mushrooms,
including immune enhancement and the treatment of a wide range of conditions and illnesses. Selected journals, references, resources, books, sources for supplements and extracts, and additional
videos are also presented.
Nordic Walking is a low-stress walking exercise that employs specially designed poles and a grip-release technique to engage the upper body in a workout similar to cross-country
skiing. Developed in Finland and officially launched in 1997, Nordic Walking is very popular in Europe. Clinical studies
have found that this technique delivers a broad range of physical and psychological benefits. Anecdotal reports include
remarkable results achieved by fitness enthusiasts as well as those suffering from chronic medical conditions. Nordic Walking
can be adapted to individual fitness levels — it isn't just for the fitness enthusiast who wants a high-intensity, total body
aerobic workout. Virtually everyone who can walk, and many who have difficulty walking, can go Nordic Walking.
The Environmental Working Group recently published a report entitled the EWG Bottled Water Scorecard 2011, in which they recommend filtered tap water purer than tap water and
preferable to the plastic bottled water alternatives, the manufacturers of which often fail to disclose contaminants and other crucial facts.
Tap or bottled, what's in your drinking water? Does the water in your shower or bath contain contaminants disinfectant byproducts (DBPs), Trihalomethanes (THMs), Volatile Organic Compounds (VOCs) and other toxins that can adversely affect your health? Can you inhale these contaminants, or absorb them through your skin?
This resource page on water quality explores water contaminants, adverse effects from the ingestion, inhalation and dermal absorption of contaminants, bottled water, water testing, water purification,
and finding the right solution to meet your personal and household needs for clean water. Selected national, state/provincial and municipal resources are provided for the United States and Canada, with an extensive list of
clinical references.
Genetically engineered crops have raised a host of issues — ethical, ecological, medical, social, legal, and
more — deeply dividing both the scientific community and the general public. GMOs have been linked to
toxins, allergies, infertility, infant mortality, immune dysfunction, stunted growth, and death. Testing is not required for foods that
fall under the GRAS (Generally Recognized As Safe) designation of the FDA, and producers effectively decide if the GM product and plant
that produces it are exempt from testing. Is there reason for concern? We believe there is, and that it's important to know what might
not be working in our best interests.
Minerals have been used for medicinal purposes, both topically and internally, for millennia. Clay minerals have been used for the treatment of wounds, skin afflictions,
stomach and intestinal problems, malaria, for cleansing, as anti-inflammatories, cicatrizers, and more. The adsorptive and absorptive properties of natural clays, with their fine
particle size and high cation exchange capacity, make them useful in a range of industrial, pharmaceutical, and cosmetic applications. They
are effective absorbers of secretions and toxins, exhibiting bacteriostatic and bactericidal properties...
Yellowfin Tuna (Thunnus albacares) Credit: US Food and Drug Administration
Regulatory Fish Encyclopedia. Adapted.
Some types of fish and seafood are safe to eat, some should be consumed in moderation, and some should be avoided altogether. There are two reasons for this. First, contaminants such as
PCBs, mercury, and other toxins in some fish and seafood poses a cumulative health risk for consumers. Second, over-fishing of certain species or groups of species has resulted in a dramatic decline in numbers.
Greenpeace reports that "[n]inety per cent of large, commercially valuable species such as cod, tuna and swordfish have vanished from the oceans, and 27 million tonnes
of marine life are caught incidentally and thrown overboard dead or dying each year". From a health perspective then, both human and marine, it is important to know which fish are safe to eat and how frequently,
as well as which fish have been obtained from well-managed sources and/or caught in an ecologically responsible manner that preserves biodiversity and habitat.
Dried Psilocybe cubensis magic mushrooms. Credit: Erik Fenderson, Wikipedia (3.12.06)
In a 2006 paper entitled Psilocybin can occasion mystical-type experiences having substantial and sustained personal meaning
and spiritual significance, authors R.R. Griffiths et al. find that volunteers administered psilocybin under supportive
conditions subjectively identify personal meaning and spiritual significance in psilocybin-occasioned mystical experiences
similar in nature to those that occur spontaneously. In a follow-up study published 1 July 2008, the authors report that, "[w]hen administered under
supportive conditions, psilocybin occasioned experiences similar to spontaneously occurring mystical experiences that, at
14-month follow-up, were considered by volunteers to be among the most personally meaningful and spiritually significant
of their lives". And see recent paper by Vollenweider & Kometer (Sep 2010): The neurobiology of psychedelic drugs: implications for the treatment of mood disorders.
William Davis, a preventive cardiologist who practises in Milwaukee, Wis., argues in his new book Wheat Belly that wheat is bad for your health — so
bad that it should carry
a surgeon general's warning. [...]
[...] Q: How does wheat make us fat, exactly?
A: It contains amylopectin A, which is more efficiently converted
to blood sugar than just about any other carbohydrate, including table sugar. In fact, two slices of whole wheat bread increase blood sugar to a higher level than a candy bar does. And then, after about two hours,
your blood sugar plunges and you get shaky, your brain feels foggy, you're hungry. So let's say you have an English muffin for breakfast. Two hours later you're starving, so you have a handful of crackers, and
then some potato chips, and your blood sugar rises again. That cycle of highs and lows just keeps going throughout the day, so you're constantly feeling hungry and constantly eating. Dieticians have responded
to this by advising that we graze throughout the day, which is just nonsense. If you eliminate wheat from your diet, you're no longer hungry between meals because you've stopped that cycle. You've cut out the
appetite stimulant, and consequently you lose weight very quickly. I've seen this with thousands of patients.
[...]In addition to the 90- to 120-minute cycle of sugar and insulin, there's the gliadin effect. Gliadin is the protein unique to wheat that stimulates appetite. Gliadin induces a subtle euphoria that
triggers a need for more, no different than an opiate like morphine, heroine, or oxycontin. Skeptical? A drug company has already made application to the FDA for the drug naltrexone, an opiate-blocking drug,
for weight loss. Does it work? You bet: 22.4 pounds lost first 6 months. NIH researchers have already shown that proteins derived from gliadin bind to opiate receptors in the brain. Skip that mid-morning
snack and gliadin withdrawal starts to kick in: shakiness, mental "fog," anxiety, accompanied by the loud rumbling rolls. [...]
Hybrid Wheat: Results and Problems. (Advances in Plant Breeding, Supplement 15.) Pickett AA. Hamburg and Berlin: Paul Parry 1993:268pp., DM96.00, ISBN 3-489-53510-3.
Hybrid wheat
Cisar G, Cooper DB. In: Curtis BC, Rajaram S, Macpherson HG (eds.), BREAD WHEAT: Improvement and Production. FAO Plant Production and Protection Series, No. 30,
Food and Agriculture Organization of the United Nations, Rome, 2002.
Q: But I'm not overweight and I exercise regularly. So why would eating whole wheat bread be bad for me?
A: You can trigger effects you don't perceive. Small low-density lipoprotein [LDL] particles form when you're eating lots of carbohydrates, and they are responsible for atherosclerotic
plaque, which in turn triggers heart disease and stroke. So even if you're a slender, vigorous, healthy person, you're still triggering the formation of small LDL particles. And second, carbohydrates
increase your blood sugars, which cause this process of glycation, that is, the glucose modification of proteins. If I glycate the proteins in my eyes, I get cataracts. If I glycate the cartilage of my
knees and hips, I get arthritis. If I glycate small LDL, I'm more prone to atherosclerosis. So it's a twofold effect. And if you don't start out slender and keep eating that fair trade, organically grown
whole wheat bread that sounds so healthy, you're repeatedly triggering high blood sugars and are going to wind up with more visceral fat. This isn't just what I call the wheat belly that you can see,
flopping over your belt, but the fat around your internal organs. And as visceral fat accumulates, you risk responses like diabetes and heart disease.
Q: You seem to be saying that aside from anything else, wheat is essentially the single cause of the obesity epidemic.
A: I wouldn't go so far as to say that all obesity is due to wheat. There are kids, of course, who drink Coca-Cola and sit in front of video games for many hours a day. But I'm speaking
to the relatively health-minded people who think they're doing the right thing by limiting fat consumption and eating more whole grains, and there's a clear subset of people who are doing that and gaining
weight and don't understand why. It causes tremendous heartache. They come into my office and say, "I exercise five times a week, I've cut my fat intake, I watch portion size and eat my whole grains — but I've
gone up three dress sizes."
Q: You write that wheat is "addictive," but does it really meet the criteria for addiction we'd use when talking about, say, drugs?
A: National Institutes of Health researchers showed that gluten-derived polypeptides can cross into the brain and bind to the brain's opiate receptors. So you get this mild euphoria
after eating a product made with whole wheat. You can block that effect [in lab animals] by administering the drug naloxone. This is the same drug that you're given if you're a heroin addict; it's an opiate
blocker. About three months ago, a drug company applied to the FDA to commercialize naltrexone, which is an oral equivalent to naloxone. And it works, apparently, it blocks the pleasurable feelings you get
from eating wheat so people stop eating so much. In clinical trials, people lost about 22.4 lb. in the first six months. Why, if you're not a drug addict, do you need something like that? And of course
there's another option, which is to cut wheat out of your diet. However, and this is another argument for classifying wheat as addictive, people can experience some pretty unpleasant withdrawal symptoms.
Q: For how long?
A: Generally about five days. And once you're through withdrawal, your cravings subside, your calorie intake decreases and your alertness and overall health improve.
Q: So do you believe food manufacturers are putting wheat into more and more food products, not just bread and crackers, because it's addictive and stimulates appetite?
A: These are not stupid people. The research showing that wheat stimulates appetite didn't come from some little alternative health practitioner. It comes from the NIH. It stretches
credibility to believe they have no awareness of the evidence.
Q: If there's all this evidence, why does the government encourage us to "eat healthy" by upping our consumption of whole grains?
A: That's the million-dollar question. Wheat is so linked to human habit, it's 20 per cent of all calories consumed by humans worldwide, that I think there was the presumption, "Gee,
humans have consumed this for thousands of years, so what's the problem?" I don't think the misguided advice to eat more whole grains came from evil intentions. [...]
[Read the full interview]
In the first article in this series, I showed that hypothyroidism is an autoimmune
disease in 90% of cases. In this article we're going to discuss the connection between autoimmune thyroid disease (AITD) and gluten intolerance.
Several studies show a strong link between AITD (both Hashimoto's and Graves') and gluten intolerance. [1,
2, 3,
4, 5]
The link is so well-established that researchers suggest all people with AITD be screened for gluten intolerance, and vice versa.
What explains the connection? It's a case of mistaken identity. The molecular structure of gliadin, the protein portion of gluten, closely resembles that of the thyroid gland. When gliadin
breaches the protective barrier of the gut, and enters the bloodstream, the immune system tags it for destruction. These antibodies to gliadin also cause the body to attack thyroid tissue. This means if you have
AITD and you eat foods containing gluten, your immune system will attack your thyroid.
Even worse, the immune response to gluten can last up to 6 months each time you eat it. This explains why it is
critical to eliminate gluten completely from your diet if you have AITD. There's no "80/20" rule when it comes to gluten. Being "mostly" gluten-free isn't going to cut it. If you're gluten
intolerant, you have to be 100% gluten-free to prevent immune destruction of your thyroid. [...]
p.31
All combined, it's estimated 43 percent of Americans are genetically predisposed to celiac disease, and 81 percent are predisposed to gluten intolerance.
When it comes to diagnosing celiac disease, other markers to consider are positive antibodies against:
Gliadin, a protein in gluten
Transglutaminase, an enzyme in the intestines
Endomysium, a muscle sheath
When any or all of these is positive, it indicates a person is not just gluten intolerant, but has celiac disease. [...]
[...I]f a person with a gluten intolerance or celiac disease eats gluten regularly, her immune system is kept on a constant
red alert, toiling virtually nonstop. Here's how it sets the stage for Hashimoto's: When immune antibodies tag gluten for removal,
they stimulate the production of antibodies against the thyroid gland as well (again, because they are both so similar in structure).
In other words, every time gluten is ingested, the immune system launches an attacknot only against gluten but also on the thyroid gland. What's worse,
the immune response to gluten can last up to six months each time it's ingested. [...] All patients with an autoimmune thyroid condition
should be screened for gluten intolerance or celiac disease,48 just as all patients with gluten intolerance or celiac disease should be screened
for an autoimmune thyroid disorder.49 [...]
Since 1995 rates have doubled or nearly doubled in 17 states.
Adult obesity rates increased in 16 states in the past year and did not decline in any state, according to F as in Fat: How Obesity Threatens
America's Future 2011, a report from Trust for America's Health (TFAH) and the Robert Wood Johnson Foundation (RWJF). Twelve states now
have obesity rates above 30 percent. Four years ago, only one state was above 30 percent.
The obesity epidemic continues to be most dramatic in the South, which includes nine of the 10 states with the highest adult obesity rates. States in
the Northeast and West tend to have lower rates. Mississippi maintained the highest adult obesity rate for the seventh year in a row, and Colorado has the
lowest obesity rate and is the only state with a rate under 20 percent.
This year, for the first time, the report examined how the obesity epidemic has grown over the past two decades. Twenty years ago, no state had an obesity
rate above 15 percent. Today, more than two out of three states, 38 total, have obesity rates over 25 percent, and just one has a rate lower than 20 percent.
Since 1995, when data was available for every state, obesity rates have doubled in seven states and increased by at least 90 percent in 10 others. Obesity rates
have grown fastest in Oklahoma, Alabama, and Tennessee, and slowest in Washington, D.C., Colorado, and Connecticut.
"Today, the state with the lowest obesity rate would have had the highest rate in 1995," said Jeff Levi, Ph.D., executive director of TFAH. "There was a
clear tipping point in our national weight gain over the last twenty years, and we can't afford to ignore the impact obesity has on our health and corresponding
health care spending."
Obesity has long been associated with other severe health problems, including diabetes and high blood pressure. New data in the report show how rates of both
also have risen dramatically over the last two decades. Since 1995, diabetes rates have doubled in eight states. Then, only four states had diabetes rates
above 6 percent. Now, 43 states have diabetes rates over 7 percent, and 32 have rates above 8 percent. Twenty years ago, 37 states had hypertension rates over
20 percent. Now, every state is over 20 percent, with nine over 30 percent.
Racial and ethnic minority adults, and those with less education or who make less money, continue to have the highest overall obesity rates:
Adult obesity rates for Blacks topped 40 percent in 15 states, 35 percent in 35 states, and 30 percent in 42 states and D.C.
Rates of adult obesity among Latinos were above 35 percent in four states (Mississippi, North Dakota, South Carolina, and Texas) and at least 30 percent in 23 states.
Meanwhile, rates of adult obesity for Whites topped 30 percent in just four states (Kentucky, Mississippi, Tennessee, and West Virginia) and no state had a rate higher than 32.1 percent.
Nearly 33 percent of adults who did not graduate high school are obese, compared with 21.5 percent of those who graduated from college or technical college.
More than 33 percent of adults who earn less than $15,000 per year were obese, compared with 24.6 percent of those who earn at least $50,000 per year.
Source: Ischemic stroke hospitalizations decline in middle aged, elderly, increases in young
American Stroke Association Meeting Report: Abstract MP70 Trends of acute ischemic stroke hospitalizations by age and gender in the United States: 1994-2007. (9.02.11).
NOTE: Published as an abstract and presented at conference, not yet published in a peer-reviewed journal. Video summary, Mary George, MD, MSPH, Division for Heart Disease and Stroke Prevention, CDC.
Study Highlights:
The prevalence of ischemic stroke has declined in the middle aged and elderly, but sharply increased in children, young adults and early middle-aged
people since 1994, according to an analysis by CDC researchers.
Researchers are unclear on the reasons for the increase, but said obesity and hypertension may be factors.
The percentage of adult Americans diagnosed with diabetes has risen steadily for the past 20 years, up to 8 percent of the population in 2008. In some regions of the country, however,
the rate is nearly twice that. Since 2004, the Centers for Disease Control and Prevention have released estimates for each county, and mapping that data
reveals that there are clusters in states like Alabama and Mississippi, where around one in seven adults is diabetic.
Click the image to visit Slate Labs and explore the maps.
LOS ANGELES, Feb. 9, 2011 — The number of acute ischemic stroke hospitalizations among middle-aged and older men and women fell between 1994 and
2007, but sharply increased among those under age 35 — including teens and children — according to research presented at the American Stroke Association's International Stroke Conference 2011.
Analysts at the U.S. Centers for Disease Control and Prevention (CDC), reviewing hospitalization data by age and gender, identified declining rates of 51 percent in girls 0-4 years and 25
percent in men and 29 percent in women over 45.
However, the number of ischemic stroke hospitalizations increased 51 percent in males between ages 15 and 34 during the period studied. The rate increased 17
percent in females between 15 and 34.
Among children and teens, they found a 31 percent increase in boys between 5 to 14 years and a 36 percent increase among girls 5 to 14 years.
Among the younger middle-aged set, they found a 47 percent increase among men 35-44 and a 36 percent increase among women 35-44.
"I believe this is the first large study to report these findings, stratified by age and gender," said Xin Tong, M.P.H., a health statistician with the CDC's
Division for Heart Disease and Stroke Prevention in Atlanta.
"We cannot link anything in particular to the trend in younger patients, but I believe the role of obesity and hypertension will prompt a big discussion.
Unfortunately, right now we can't speculate on the causes."
The unit of analysis was hospitalization, so researchers couldn't draw any firm connections or determine what factors are driving the increase in ischemic
stroke cases among the young. Ischemic stroke occurs when blood supply to the brain becomes obstructed, usually by a clot or narrowing of the arteries. The
risk of long-term brain damage can be reduced significantly if patients receive the clot-busting tissue plasminogen activator (tPA) within three or four and a
half hours after stroke onset.
Hospitals and physicians should be aware of the rising risk of stroke in young people, and the necessity to educate them about stroke symptoms, Tong said.
"Acute ischemic stroke is currently considered something that mostly happens to older people, but awareness of rising rates in the young is important or else
tPA and other important stroke treatment may be unnecessarily delayed in younger patients," she said. [...]
The above study does not tie risk factors to trend findings.
For identification of risk factors, see instead:
ROCHESTER, Minn. — One of the largest studies of its kind has found that people with coronary artery disease who have even a modest beer belly or
muffin top are at higher risk for death than people whose fat collects elsewhere. The effect was observed even in patients with a normal Body Mass
Index (BMI). The findings of this Mayo Clinic analysis are published in the May 10 issue of the Journal of the American College of Cardiology.
Researchers analyzed data from 15,923 people with coronary artery disease involved in five studies from around the world. They found that those with
coronary artery disease and central obesity, measured by waist circumference and waist-to-hip ratio, have up to twice the risk of dying. That is
equivalent to the risk of smoking a pack of cigarettes per day or having very high cholesterol, particularly for men.
The findings refute the obesity paradox, a puzzling finding in many studies that shows that patients with a higher BMI and chronic diseases such as
coronary artery disease have better survival odds than normal-weight individuals.
Central obesity is measured by comparing the circumference of a patient's stomach to the circumference of the hips. When the stomach measurement is 90 percent or more of the hip
measurement in men and 85 percent or more of the hip measurement in women a patient generally is thought to have a worrisome distribution of fat, said Dr. Francisco Lopez-Jimenez,
director of the cardiometabolic program at the Mayo Clinic.
"We suspected that the obesity paradox was happening because BMI is not a good measure of body fatness and gives no insight into the distribution of fat," says
Thais Coutinho, M.D., the study's lead author and a cardiology fellow at Mayo Clinic. "BMI is just a measure of weight in proportion to height. What seems
to be more important is how the fat is distributed on the body," she says.
Francisco Lopez-Jimenez, M.D., the project's lead investigator and director of the Cardiometabolic Program at Mayo Clinic, explains why this type of fat may be
more harmful: "Visceral fat has been found to be more metabolically active. It produces more changes in cholesterol, blood pressure and blood sugar. However,
people who have fat mostly in other locations in the body, specifically, the legs and buttocks, don't show this increased risk."
The researchers say physicians should counsel coronary artery disease patients who have normal BMIs to lose weight if they have a large waist circumference
or a high waist-to-hip ratio. The measure is very easy to use, Dr. Coutinho says: "All it takes is a tape measure and one minute of a physician's time to
measure the perimeter of a patient's waist and hip."
The research subjects were diverse, coming from studies in the U.S. (Rochester, Minn. and San Francisco, Calif.), Denmark, France and Korea. The inclusion
of different ethnic groups makes the study more applicable to the real world, Dr. Coutinho says. [...]
The vital interplay of D and K in bone and heart health
D and K: A Vital Interplay in Bone and Heart Health
In all likelihood, your cardiologist is unaware that the mechanisms underlying arterial calcification closely resemble the process of new bone
formation, involving many of the same cells (including osteoblasts), proteins, and cytokines (signaling molecules).9
Vitamin K2 Contents of Selected Foods1, 2
The percentage of vitamin K2 present as MK-4 represents that synthesized by animal tissues, while the remainder represents that synthesized
by bacteria during fermentation.
Vitamin K is obtained from the diet as phylloquinone (vitamin K-1) and menaquinone (MK-n, vitamin K-2). Phylloquinone is abundant in dark-green leafy vegetables and vegetable oils. The main
dietary sources for menaquinone in Western populations are meats (MK-4) and fermented foods, especially cheese and curds (mainly MK-8 and MK-9).
MK-7 and MK-8,9,10 come from bacterial fermentation, whether in natto, cheese, or in your
intestinal tract; MK-4 is naturally synthesized by animals from vitamin K1. While natto is the richest source of the MK-7 form, egg yolks
and fermented cheeses are the richest sources of the MK-4 form.
Chicken contains about 8 mcg MK-4 per 3 1/2 oz serving; beef contains about 1 mcg. Egg yolks contain 31 mcg MK-4 per 3 1/2 oz serving (app. 6 raw yolks). Hard cheeses contain about 5 mcg
MK-4 per 3 1/2 oz serving, about 70 mcg of MK-8,9; soft cheeses contain about 30% less. Nattō contains about 1000 mcg of MK-7, 84 mcg MK-8, and no MK-4 per 3 1/2 oz serving.
Vitamin K2 supplementation (via frequent natto consumption or pharmaceutical doses of K2) substantially improves bone health. While K2 by itself exerts significant bone density/strength increasing properties
in dozens of studies, when combined with other bone health-promoting agents (e.g., vitamin D3, prescription drugs like Fosamax and calcitonin), an exaggerated synergy of bone health-promoting effects develop.
The MK-4 form of vitamin K2 is short-lived, lasting only 3-4 hours in the body. The MK-7 form, in contrast, the form in natto, lasts several days. MK-7 and MK-8-10 are extremely well absorbed, virtually complete.
Bone health benefits have been shown for both the MK-7 and MK-4 forms. [...]
People with osteoporosis are more likely to exhibit atherosclerotic calcification in their blood vessels. And those with atherosclerosis are more
likely to possess lower bone mass. What do these groups have in common? Both exhibit insufficient vitamin K levels.5-8
Researchers have since delineated the complex process by which the body manages calcium uptake, distribution, and deposition.5 Many of the same factors that regulate
healthy calcium levels in bones are also implicated in the destructive accumulation of calcium in arteries.10 Among those factors are specific proteins called
Gla proteins, found in bone tissue and in vascular walls, that require vitamin K for their proper function.5
Other factors crucial to atherosclerosis and osteoporosis prevention are modulated by vitamin D. These include fat-derived inflammatory cytokines.5,11
Osteoporosis and atherosclerosis, in other words, both involve insufficiencies of D and K.
Vitamin D
Vitamin D's role in the formation and maintenance of healthy bone structure and function has been established for decades. It is a vital co-factor in bone
mineralization through the absorption of calcium and phosphorus. Severe D deficiency may thus lead to rickets, a childhood disease characterized by
impeded growth and deformity of the long bones of the body.
More recently, its definitive importance in optimizing cardiovascular health has emerged. Vitamin D inhibits vascular calcification by blocking the
release of inflammatory cytokines and adhesion molecules and preventing abnormal changes in smooth muscle cells in vessel walls.13 Accordingly, low vitamin D levels are
associated with increased risk for development of the coronary arterial calcifications seen in atherosclerosis.14
Vitamin D deficiency is also associated with multiple risk factors for cardiovascular disease — including hypertension, diabetes, increased carotid artery intima-media thickness, as well as heart
attack and stroke.15 Vitamin D also reduces gene expression of bone-forming cells abnormally present in the aortas of experimental animals with
chronic kidney disease.16
A 2009 national health survey found "a strong and independent relationship of vitamin D deficiency with prevalent cardiovascular disease in a
large sample representative of the US adult population."17 Low vitamin D levels have also been implicated in congestive heart failure (CHF).18
Replenishment of low vitamin D levels provides a simple and effective means of reversing many of these risks. To take one example, a 2009 study examined the
effect of monthly injections of 300,000 IU of vitamin D3 in a group of deficient subjects with no overt symptoms of cardiovascular disease.11 At the outset of the study,
subjects had low flow-mediated dilation of their arteries, a key index of endothelial health. After only 3 months of supplementation, significant improvement in flow-mediated dilation was
observed, with diminished post-treatment measures of oxidative stress as well.
These findings have been complemented by recent research into the mechanisms dependent upon vitamin K for optimal heart and bone health—mechanisms that
operate both parallel to and in tandem with vitamin D.
Vitamin K
Vitamin K is not a single nutrient, but rather denotes several related nutritional compounds. These can be produced within the human body but not by
the body.19 Gut flora (beneficial intestinal bacteria) generate about 75% of the vitamin K your body absorbs each
day, with the other 25% coming from dietary sources.20 Just as importantly, vitamin K is not stored
in the body, underscoring the need for daily intake.21
A traditional Japanese food made from fermented soybeans, Nattō is a very rich source of K2. Image Credit: Wikipedia.
It occurs in nature in two primary forms: K1 or phylloquinone and K2 or menaquinone. Vitamin K is a cofactor required to convert the amino
acid glutamate into gamma-carboxyglutamate, or Gla-proteins.22 Gla-proteins regulate physiological processes controlled by
calcium. These include blood coagulation (clotting) and bone mineralization.
Accordingly, Gla-proteins are critical to the formation and replenishment of bone tissue. Unless these proteins are modified by vitamin K, they cannot
properly form the matrix in which calcium and phosphorus bind together to make solid, well-mineralized bone. Vitamin K has been shown to stimulate new bone
formation and reduce the incidence of vertebral fractures.23,24
The Gla-protein osteocalcin, normally present in bone, has been found in calcified atherosclerotic plaque lesions, and production of this protein is pathologically upregulated in people with
atherosclerosis.25-28
At the same time, another vitamin K-dependent Gla-protein known as MGP (for "matrix Gla-protein"), normally found in healthy arterial walls, is a strong inhibitor of vascular
calcification.29,30 In other words, by increasing matrix Gla-protein in the arterial walls, vitamin K protects against the calcification-inducing effects of
osteocalcin.
Super K with Advanced K2 Complex
Swanson Ultra Nattō K-7 Natural Vitamin K-2
This may explain the emergence of compelling evidence for vitamin K as a key factor in overall heart health. To take one example, a large study of more than
4,800 subjects followed for 7-10 years in the Netherlands demonstrated that people in the highest one-third of vitamin K2 intake had a
57% reduction in risk of dying from cardiovascular disease, compared to those with the lowest intake. And their risk of having severe aortic
calcification plummeted by 52% — a clear demonstration of the vitamin's protective effects.31 Another
study by the same group showed that vitamin K2 intake was associated with a 20% decreased risk of coronary artery
calcification.32 [...]
Tintut Y, Demer LL. Recent advances in multifactorial regulation of vascular calcification. Curr Opin Lipidol. 2001 Oct;12(5):555-60.
Jie KS, Bots ML, Vermeer C, Witteman JC, Grobbee DE. Vitamin K intake and osteocalcin levels in women with and without aortic atherosclerosis: a population-based study. Atherosclerosis. 1995 Jul;116(1):117-23.
Jie KG, Bots ML, Vermeer C, Witteman JC, Grobbee DE. Vitamin K status and bone mass in women with and without aortic atherosclerosis: a population-based study. Calcif Tissue Int. 1996 Nov;59(5):352-6.
Hmamouchi I, Allali F, Khazzani H, et al. Low bone mineral density is related to atherosclerosis in postmenopausal Moroccan women. BMC Public Health. 2009;9:388.
Danilevicius CF, Lopes JB, Pereira RM. Bone metabolism and vascular calcification. Braz J Med Biol Res. 2007 Apr;40(4):435-42.
Tintut Y, Morony S, Demer LL. Hyperlipidemia promotes osteoclastic potential of bone marrow cells ex vivo. Arterioscler Thromb Vasc Biol. 2004 Feb;24(2):e6-10.
Maetani M, Maskarinec G, Franke AA, Cooney RV. Association of leptin, 25-hydroxyvitamin D, and parathyroid hormone in women. Nutr Cancer. 2009;61(2):225-31.
Calcification and osteoporosis from clinical observation towards molecular understanding. Osteoporos Int. 2007 Mar;18(3):251-9.
Zittermann A, Schleithoff SS, Koerfer R. Vitamin D and vascular calcification. Curr Opin Lipidol. 2007 Feb;18(1):41-6.
de Boer IH, Kestenbaum B, Shoben AB, Michos ED, Sarnak MJ, Siscovick DS. 25-hydroxyvitamin D levels inversely associate with risk for developing coronary artery calcification. J Am Soc Nephrol. 2009 Aug;20(8):1805-12.
Gouni-Berthold I, Krone W, Berthold HK. Vitamin D and cardiovascular disease. Curr Vasc Pharmacol. 2009 Jul;7(3):414-22.
Mathew S, Lund RJ, Chaudhary LR, Geurs T, Hruska KA. Vitamin D receptor activators can protect against vascular calcification. J Am Soc Nephrol. 2008 Aug;19(8):1509-19.
Kendrick J, Targher G, Smits G, Chonchol M. 25-Hydroxyvitamin D deficiency is independently associated with cardiovascular disease in the Third National Health and Nutrition Examination Survey. Atherosclerosis. 2009 Jul;205(1):255-60.
Szabo B, Merkely B, Takacs I. The role of vitamin D in the development of cardiac failure. Orv Hetil. 2009 Jul 26;150(30):1397-402.
Mueller RL, Scheidt S. History of drugs for thrombotic disease. Discovery, development, and directions for the future. Circulation. 1994 Jan;89(1):432-49.
Miggiano GA, Robilotta L. Vitamin K-controlled diet: problems and prospects. Clin Ter. 2005 Jan-Apr;156(1-2):41-6.
Israels LG, Israels ED, Saxena SP. The riddle of vitamin K1 deficit in the newborn. Semin Perinatol. 1997 Feb;21(1):90-6.
Askim M. Vitamin K in the Norwegian diet and osteoporosis. Tidsskr Nor Laegeforen. 2001 Sep 20;121(22):2614-6.
Iwamoto J, Takeda T, Sato Y. Effects of vitamin K2 on osteoporosis. Curr Pharm Des. 2004;10(21):2557-76.
Iwamoto J, Takeda T, Sato Y. Role of vitamin K2 in the treatment of postmenopausal osteoporosis. Curr Drug Saf. 2006 Jan;1(1):87-97.
Vermeer C, Shearer MJ, Zittermann A, et al. Beyond deficiency: potential benefits of increased intakes of vitamin K for bone and vascular health. Eur J Nutr. 2004 Dec;43(6):325-35.
Braam LA, Dissel P, Gijsbers BL, et al. Assay for human matrix gla protein in serum: potential applications in the cardiovascular field.
Arterioscler Thromb Vasc Biol. 2000 May;20(5):1257-61.
Shanahan CM, Proudfoot D, Farzaneh-Far A, Weissberg PL. The role of Gla proteins in vascular calcification. Crit Rev Eukaryot Gene Expr. 1998;8(3-4):357-75.
Levy RJ, Gundberg C, Scheinman R. The identification of the vitamin K-dependent bone protein osteocalcin as one of the gamma-carboxyglutamic acid
containing proteins present in calcified atherosclerotic plaque and mineralized heart valves. Atherosclerosis. 1983 Jan;46(1):49-56.
Schurgers LJ, Dissel PE, Spronk HM, et al. Role of vitamin K and vitamin K-dependent proteins in vascular calcification. Z Kardiol. 2001;90 Suppl 3:57-63.
Shoji S. Vitamin K and vascular calcification. Clin Calcium. 2002 Aug;12(8):1123-8.
Let thy Food be thy Medicine and thy Medicine be thy Food - Hippocrates.
Food Matters is a hard hitting, fast paced look at our current state
of health. Despite the billions of dollars of funding and research into new so-called cures we continue to suffer from a raft of chronic ills and every day maladies.
The film sets about uncovering the trillion dollar worldwide 'Sickness Industry' and exposes a growing body of scientific evidence proving that
nutritional therapy can be more effective, more economical, less harmful and less invasive than most conventional medical treatments.
Food Matters features interviews with leading medical experts from around the world who
discuss natural approaches to preventing and reversing Cancer, Obesity, Heart Disease, Depression, Mental Illness and many other chronic conditions.
Find out what works, what doesn't and what's killing you. Becoming informed about the choices you have for you and your family's health could save your life.
The Food Matters Store features a collection of must-watch films on Nutrition, Natural Healing and Sustainability. Browse through the
titles and watch the trailers of these excellent documentaries and how-to films.
Washington, DC - Environmental Working Group has released the seventh edition of its Shopper's Guide to Pesticides in Produce with updated information on 53
fruits and vegetables and their total pesticide loads. EWG highlights the worst offenders with its "Dirty Dozen" list and the cleanest conventional produce with its "Clean 15" list.
Analysts at EWG synthesized data collected from the U.S. Department of Agriculture and the Food and Drug Administration from 2000 to 2009. Produce is
ranked based on a composite score, equally weighing six factors that reflect how many pesticides was found in testing of on each type of the produce and at what
levels. Most samples are washed and peeled prior to being tested, so the rankings reflect the amounts of the chemicals likely present on the food when is it eaten.
Notable changes in the new guide included apples' rank as the most contaminated produce, jumping three spots from last year to replace celery at
the top of the "Dirty Dozen" list. According to USDA, pesticides showed up on 98 percent of the more than 700 apple samples tested.
Making an appearance in the guide for the first time is the herb cilantro, which had never been tested by USDA until now. The data showed 33 unapproved
pesticides on 44 percent of the cilantro samples tested, which is the highest percentage of unapproved pesticides recorded on any item included in the guide since EWG started tracking the data in 1995.
Also appearing in the guide for the first time are green onions, cranberries and mushrooms. Mushrooms made the "Clean 15" list, while honeydew was the only
item to drop off that list this year. Cherries dropped off the "Dirty Dozen" list, but lettuce, which has made the list in previous years, were back on.
"Though buying organic is always the best choice, we know that sometimes people do not have access to that produce or cannot afford it," said EWG
President Ken Cook. "Our guide helps consumers concerned about pesticides to make better choices among conventional produce, and lets them know which fruits
and vegetables they may want to buy organic."
Pesticides can be extremely toxic to human health and the environment. U.S. and international government agencies alike have linked pesticides to nervous
system toxicity, cancer, hormone system disruption and IQ deficits among children.
"I really worry that pesticides on food are unhealthy for the tender, developing brains and bodies of young children," said Dr. Harvey Karp, MD, FAAP,
creator of the book/DVD The Happiest Baby on the Block. "Parents don't realize they're often feeding their little ones fruits and veggies with the highest
pesticide residues. Studies show even small amounts of these chemicals add up and can impair a child's health when they're exposed during the early, critical
stages of their development. When pesticide sprayers have to bundle up in astronaut-like suits for protection, it's clear parents want to feed their
families food containing as little of these toxic chemicals as possible."
"Pesticides, while designed specifically to kill certain organisms, are also associated with a host of very serious health problems in people, including
neurological deficits, ADHD, endocrine system disruption and cancer," said Andrew Weil, MD, Founder and Director, Arizona Center for Integrative Medicine
and a renowned medical expert on natural health and wellness. "My advice to consumers is to whenever possible avoid exposure to pesticides, including
pesticide residues on food."
Consumers who choose five servings of fruits and vegetables a day from EWG's Clean 15 list rather than from the Dirty Dozen can lower the volume of
pesticides they consume by 92 percent, according to EWG's calculations. They will also eat fewer types of pesticides. Picking five servings of fruits and
vegetables from the 12 most-contaminated products would result in consuming an average of 14 different pesticides a day. Choosing five servings from the 15
least contaminated fruits and vegetables would result in consuming fewer than two pesticides per day.
The health benefits of a diet rich in fruits and vegetables outweigh the risks of pesticide exposure, and EWG strongly recommends that everyone follow
USDA's recommendation to eat five servings of fruits and vegetables every day. EWG's Shopper's Guide makes it easy to meet that goal while reducing your exposure to pesticides.
"Pesticides are toxic," said Sonya Lunder, Senior Analyst at EWG. "They are designed to kill things and most are not good for you. The question is, how bad are they?"
EWG's Shoppers Guide is available for fee as a PDF download at http://www.ewg.org/foodnews/. An iPhone
app will be available in the near future. For a small donation, consumers can also have a version of the guide sent to them as a bag tag that can be attached to reusable shopping bags.
EWQ notes that [t]he data used to create the Shopper's Guide are from produce tested as it is typically eaten. This means washed and,
when applicable, peeled. For example, bananas are peeled before testing, and blueberries and peaches are washed. Because all produce has been thoroughly cleaned before analysis, washing a fruit or
vegetable would not change its rank in the EWG's Shopper's Guide. Remember, if you don't wash conventional produce, the risk of ingesting pesticides is even greater than reflected by USDA test data.
EWG has not evaluated various produce washes for efficacy or potentially toxicity. However, since some plants absorbed pesticides systemically, a produce wash would have limited effect. The safest
choice is to use the Shopper's Guide to avoid conventional versions of those fruits and vegetables with the highest pesticide residues.
Rinsing may reduce but does not eliminate pesticides; however, a more thorough cleaning may help remove topical impurities to which fruit or produce has been exposed in the journey from producer
to consumer. Here are a few examples of popular products and approaches.
[...] "We are not claiming that bilingualism in any way prevents Alzheimer's or other dementias, but it may contribute to cognitive reserve in the brain which
appears to delay the onset of Alzheimer's symptoms for quite some time," said Dr. Craik, lead investigator and co-editor of The Oxford Handbook of Memory.
The brains of people who speak two languages still show deterioration from Alzheimer's pathology; however, their special ability with two languages seems
to equip them with compensatory skills to hold back the tell-tale symptoms of Alzheimer's, such as memory loss, confusion, and difficulties with
problem-solving and planning.
"These results are especially important for multicultural societies like ours in Canada where bilingualism is common," said Dr. Bialystok, professor of
Psychology at York University and associate scientist at the Rotman Research Institute. "We need to understand how bilingualism changes cognitive ability,
especially when there are clinical implications as in this case." [...]
Objectives: There is strong epidemiologic evidence to suggest that older adults who maintain an active lifestyle in terms of social, mental, and physical engagement are protected to some degree
against the onset of dementia. Such factors are said to contribute to cognitive reserve, which acts to compensate for the accumulation of amyloid and other brain pathologies. We present evidence that
lifelong bilingualism is a further factor contributing to cognitive reserve.
Methods: Data were collected from 211 consecutive patients diagnosed with probable Alzheimer disease (AD). Patients' age at onset of cognitive impairment was recorded, as was information on
occupational history, education, and language history, including fluency in English and any other languages. Following this procedure, 102 patients were classified as bilingual and 109 as monolingual.
Results: We found that the bilingual patients had been diagnosed 4.3 years later and had reported the onset of symptoms 5.1 years later than the monolingual patients. The groups were equivalent on
measures of cognitive and occupational level, there was no apparent effect of immigration status, and the monolingual patients had received more formal education. There were no gender differences.
Conclusions: The present data confirm results from an earlier study, and thus we conclude that lifelong bilingualism confers protection against the onset of AD. The effect does not appear to be
attributable to such possible confounding factors as education, occupational status, or immigration. Bilingualism thus appears to contribute to cognitive reserve, which acts to compensate for the effects
of accumulated neuropathology.
Abstract
A recent paper by Bialystok et al in Neuropsychologia (vol. 45, pgs. 459 to 464) suggested that early bilingualism produced a statistically significant 4.1-year delay in onset of memory loss symptoms in
older individuals with Alzheimer disease, possibly reflecting an increase in the cognitive reserve of these individuals. That study focused on multilingual elderly patients of whom 90% were immigrants.
Our memory clinic, in Montreal Canada, has the advantage of having a large set of individuals who are either multilingual immigrants to Canada, or who are nonimmigrants but raised in both official languages
of Canada—French and English. We thus attempted to replicate the above findings using a larger cohort in a different setting. We examined age at diagnosis of Alzheimer disease and age at symptom onset for all
unilingual versus multilingual participants, and then for those who were nonimmigrant English/French bilinguals. Overall, we found a small but significant protective effect of more than 2 languages spoken, but
we found no significant benefit in bilinguals overall in relation to age at diagnosis or age at symptom onset. However, in the immigrant group, the results mirrored those of Bialystok et al with 2 or more
languages delaying the diagnosis of Alzheimer disease by almost 5 years. A trend toward the same effect was also seen in nonimmigrants whose first language was French. In contrast, in nonimmigrants whose first
language was English, no such effect was found. These results are discussed in relation to the earlier findings and the theory of cognitive reserve.
On Thursday, 12 November 2009, CBC Ideas ran an interview with Sue Johnson EdD, an internationally recognized couples therapist. The interview explored the importance of secure
attachment in the formation and maintenance of lasting relationships. Dr. Johnson spoke at length about the science of love, and emphasized the pioneering work on attachment theory by
John Bowlby (1907-1990), a British psychiatrist and psychoanalyst.
Hold Me Tight provides a streamlined version of EFT, walking the reader through seven conversations that capture the defining moments in a love relationship, showing how to shape these moments to create a secure and lasting bond.
Case histories and exercises in each conversation bring the lessons of EFT to life1.
Sue Johnson EdD is Professor of Clinical Psychology at The University of Ottawa, Director of the Ottawa Couple and Family Institute Inc. and the International Center for Excellence in Emotionally Focused Therapy.
She is a member of the editorial board of the Journal of Marital and Family Therapy, the Journal of Couple and Relationship Therapy, and the Journal of Family Psychology. She is a Distinguished Research Professor in
the Marital & Family Therapy Program, Alliant University, San Diego.
The following excerpt is drawn from My How Couples Therapy has Changed! Attachment, Love and Science (Sue Johnson EdD, psychotherapy.net, 2008).
Two podcasts follow in the first, CBC's Shiela Rogers interviews Sue Johnson on "Hold Me Tight"; the second is the interview from CBC Ideas, a CD version of which may be puchased at The CBC Shop.
Emphasis in original..
[...] Emotionally Focused Couple Therapy (EFT) [is] a systematic, rigorous, tested set of interventions based on the attachment
view of love and bonding. I recently summarized attachment based approaches in a manner that can be offered to clients and the public in
Hold Me Tight: Seven Conversations for a Lifetime of Love. The great strength of this new
scientific perspective is exactly that it offers a rigorous body of observation and research into what love is all about and how it changes shape and color. More so, it is a tested approach to
intervention with excellent outcome data and clinical relevance. Clients also tell us that this way of seeing and working does indeed go to the heart of the matter. In this article I will summarize
the attachment perspective and how it is supported by different strands of relationship science (these science strands will be in italics to find or avoid, as you wish!) and how it translates into
practice in EFT.
A new scientific and practical theory of love
The multitude of studies on adult attachment that have emerged over the last decade tell us that the essence of love is not a negotiated exchange of resources (so why teach negotiation skills?),
a friendship, Nature's trick to get you to mate and pass on your genes, or a time-limited episode of delusional addiction. Love is a very special kind of emotional bond, the need for which is
wired into our brain by millions of years of evolution. It is a survival imperative. The human brain codes isolation and abandonment as danger and the touch and emotional responsiveness of loved
ones as safety, a safety that promotes optimal flexibility and continual learning. Jaak Panksepp, in his neurobiological studies finds that loss of connection from attachment figures triggers
"primal panic," a special set of fear responses. As Bowlby notes the words anxiety and anger come from the same etymological root and both arise at moments of disconnection, when attachment
figures are non-responsive. This need for emotional connection is not a sentimental notion. The basic image of who we are and what our most basic needs are, namely, that we are social animals
who seek such connection is reflected in health studies. For example, it is now clear that emotional isolation is more dangerous for your health than smoking and that it doubles the likelihood
of heart attack and stroke.
Love is a very special kind of emotional bond, the need for which is wired into our brain by millions of years of evolution. It is a survival imperative.
Attachment theory states that we need a safe haven relationship to turn to when life is too much for us and that offers us a secure base from which to go confidently out into the world.
This is effective dependency. Many psychotherapy clients learn that their problem is that they are too close or undifferentiated from loved ones. The approach discussed here offers a larger
picture. The evidence is that secure close connection is a source of strength and personality integration rather than weakness. Studies show that the securely connected have a more articulated
and positive sense of self. Eighteen months after 9/11, researcher Chris Fraley found that securely connected survivors, who could turn to others for emotional support, were able to deal
with this trauma and grow from it, whereas insecurely attached survivors were experiencing significant mental health problems. Secure connection is shaped by mutual emotional accessibility
and responsiveness. This is the heart of the drama that plays out in the couple therapist's office. The fights that matter in a relationship are only superficially about the kids or money.
Partners and therapists can spend many hours talking about these content issues instead of focusing on how the couple talk and more specifically, on the key attachment questions that drive
a couples negative dance. The key questions are: "Are you there for me?" "Do I matter to you?" "Will you turn towards me and respond to me?" Partners often do not know how to ask these questions
and therapists often miss them or even see them as a sign of immature dependency. [...] [Read in full]
The Next Chapter Podcast (23.05.09)
Shiela Rogers interviews Sue Johnson on "Hold Me Tight"
Ideas Podcast (12.11.09) CBC Ideas producer Richard Handler interviews Sue Johnson The independent, autonomous self is lionized in our culture. But recognizing the hold that attachment has on us, is the secret of lasting relationships. So says Sue Johnson,
a leading couples' therapist and a Canadian with an international following... Listen Now
Publication of the fifth edition of Diagnostic and Statistical Manual of Mental Disorders (DSM-5) in May 2013 will mark one the most anticipated events in the mental health field.
As part of the development process, the preliminary draft revisions to the current diagnostic criteria for psychiatric diagnoses are now available for public review. We thank you
for your interest in DSM-5 and hope that you use this opportunity not only to learn more about the proposed changes in DSM-5, but also about its history, its impact, and its developers.
Please continue to check this site for updates to criteria and for more information about the development process.
As chairman of the task force that created the current Diagnostic and Statistical Manual of Mental Disorders (DSM-IV), which came out in 1994, I learned from painful experience how small changes
in the definition of mental disorders can create huge, unintended consequences.
Our panel tried hard to be conservative and careful but inadvertently contributed to three false "epidemics" -- attention deficit disorder, autism and childhood bipolar disorder. Clearly, our net
was cast too wide and captured many "patients" who might have been far better off never entering the mental health system.
The first draft of the next edition of the DSM, posted for comment with much fanfare last month, is filled with suggestions that would multiply our mistakes and extend the reach of psychiatry dramatically
deeper into the ever-shrinking domain of the normal. This wholesale medical imperialization of normality could potentially create tens of millions of innocent bystanders who would be mislabeled as having a
mental disorder. The pharmaceutical industry would have a field day -- despite the lack of solid evidence of any effective treatments for these newly proposed diagnoses.
The manual, prepared by the American Psychiatric Assn., is psychiatry's only official way of deciding who has a "mental disorder" and who is "normal." The quotes are necessary because this distinction is
very hard to make at the fuzzy boundary between the two. If requirements for diagnosing a mental disorder are too stringent, some who need help will be left out; but if they are too loose, normal people
will receive unnecessary, expensive and sometimes quite harmful treatment.
Where the DSM-versus-normality boundary is drawn also influences insurance coverage, eligibility for disability and services, and legal status -- to say nothing of stigma and the individual's sense of personal
control and responsibility.
What are some of the most egregious invasions of normality suggested for DSM-V? "Binge eating disorder" is defined as one eating binge per week for three months. (Full disclosure: I, along with more than 6%
of the population, would qualify.) "Minor neurocognitive disorder" would capture many people with no more than the expected memory problems of aging. Grieving after the loss of a loved one could frequently be
misread as "major depression." "Mixed anxiety depression" is defined by commonplace symptoms difficult to distinguish from the emotional pains of everyday life.
The recklessly expansive suggestions go on and on. "Attention deficit disorder" would become much more prevalent in adults, encouraging the already rampant use of stimulants for performance enhancement.
The "psychosis risk syndrome" would use the presence of strange thinking to predict who would later have a full-blown psychotic episode. But the prediction would be wrong at least three or four times
for every time it is correct -- and many misidentified teenagers would receive medications that can cause enormous weight gain, diabetes and shortened life expectancy.
A new category for temper problems could wind up capturing kids with normal tantrums. "Autistic spectrum disorder" probably would expand to encompass every eccentricity. Binge drinkers would be
labeled addicts and "behavioral addiction" would be recognized. (If we have "pathological gambling," can addiction to the Internet be far behind?)
The sexual disorders section is particularly adventurous. "Hypersexuality disorder" would bring great comfort to philanderers wishing to hide the motivation for their exploits behind a psychiatric excuse. "Paraphilic
coercive disorder" introduces the novel and dangerous idea that rapists merit a diagnosis of mental disorder if they get special sexual excitement from raping.
Defining the elusive line between mental disorder and normality is not simply a scientific question that can be left in the hands of the experts. The scientific literature is usually limited, never easy to
generalize to the real world and always subject to differing interpretations.
Experts have an almost universal tendency to expand their own favorite disorders: Not, as alleged, because of conflicts of interest -- for example, to help drug companies, create new customers or increase
research funding -- but rather from a genuine desire to avoid missing suitable patients who might benefit. Unfortunately, this therapeutic zeal creates an enormous blind spot to the great risks that come
with overdiagnosis and unnecessary treatment.
This is a societal issue that transcends psychiatry. It is not too late to save normality from DSM-V if the greater public interest is factored into the necessary risk/benefit analyses.
Allen Frances MD, currently professor emeritus at Duke, was chair of the DSM-IV Task Force and of the department of psychiatry at Duke University School of Medicine, Durham, NC.
He has recently published a series of critical commen- taries on the DSM revision process see, e.g.:
For more on the DSM-5, visit DSM - Psychiatric Times (registration may be required, but there is no charge).
Your pain is the breaking of the shell that encloses your understanding.
— Kahlil Gibran (1883–1931) On Pain.
Hearing Voices...
Source:Welcome Message
Prof. Marius Romme MD PhD, President, INTERVOICE: The International Community for Hearing Voices. Accessed 8.01.11
In 1987, I had no idea the impact that the discovery that accepting and making sense of voices was a helpful alternative was going to have. Yet, after
twenty three years of work we have built a unique and formidable movement of voice hearers and allies that has brought about a big change in the way hearing
voices are regarded and has found new ways of helping people overwhelmed by their voices.
There are many fears and misunderstandings in society and within psychiatry about hearing voices. They are generally regarded as a symptom of an illness,
something that is negative, to be got rid of and consequently the content and meaning of the voice experience is rarely discussed.
The research of Dr. Sandra Escher and myself with over 300 voice hearers has shown that over 70% of people who hear voices can point to a traumatic life
event that triggered their voices; that talking about voices and what they mean is a very effective way to reduce anxiety and isolation; and that even when the
voices are overwhelming and seemingly destructive they often have an important message for the hearer.
Typically, in Western medical thinking hearing voices has always been associated with mental illness and frequently seen as a symptom of schizophrenia. Yet, we discovered many people who hear voices do not have a
mental illness and never seek help. For this reason we are prepared to accept a range of explanations offered by people who hear voices, including spiritual
ones, and believe it is essential to the process of recovery from overwhelming voices to understand the meaning of the voices to the voice hearer.
Whilst we are finding more holistic solutions to voices that cause mental distress then those offered by psychiatry. It is very important to stress that
in our view voices are an aspect of human differentness, rather than a mental health problem. As with homosexuality, which was also regarded by psychiatry in
recent times as an illness, the main issue we have to confront is the denial of the human rights to people who hear voices and our main task is to change the
way society perceives the experience. Only if can we do this, do we believe psychiatry will change its mind about voices. That is why this website is so important.
With the support of the worldwide hearing voices network, voice hearers, some of whom have spent long periods of time in psychiatric care, have reclaimed
their lives and are now able to say they hear voices and accept them as part of themselves. We believe this is a good time to make our work better known across the world.
HEARING VOICES: A Common Human Experience
John Watkins (2008:316pp)
Although hearing voices is often considered a hallmark of madness it is actually a rather common experience. While "voices" are a prominent symptom of psychotic disorders like schizophrenia
they can, also, occur in many other contexts. Many well-adjusted individuals have had at least one memorable voice experience and some people have them regularly. While some experiences are
disturbing, others provide comfort, reassurance and guidance. Benign inner voices often occur in association with non-ordinary states of consciousness, mystical and paranormal phenomena, near-death
experiences and shamanic practices and may serve as a vehicle for creative inspiration, extrasensory communication, the call of vocation and spiritual revelation. HEARING VOICES ventures beyond
conventional psychiatric therapies whose sole aim is symptom eradication to explore ways of working creatively with voices and other inner experiences to foster personal growth, healing and recovery.
Included is: - A detailed description of a wide variety of voice hearing experiences - An overview of the theories accounting for how and why this happens - A range of practical techniques for coping
with voices - Guidelines for applying spiritual discernment to hearing voices - Strategies for optimising the personal value of voice hearing experiences. deep books ltd
Intraspec.ca : Tools for Personal Development Readings, writings and research on matters of health and well-being. Richard Dagan. 2002-2012. A not-for-profit site.
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Exceptions, where copyright resides with the original author or current holder thereof, include all excerpted text and third-party images provided herein. All such material is presented in accord with
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